Inflammatory condition induced by the ingestion of gluten (protein found in wheat, barley and rye)
Aetiology
- Genetically linked auto-immune disorder
- Genetic susceptibility + trigger (trigger currently unknown, possibly enteric virus)
- HLADQ2/HLADQ8 gene - code for type II MHC molecules
- Average age of diagnosis - 40-60 years
Pathophysiology
- Not an allergy - T cell/IEL mediated
- Activated T cells and IELs kill epithelial cells
- Causes damage to small intestine, leading to malnutrition
- Damage is reversible (usually) - damaged enterocytes are replaced by normal ones when patients stop eating gluten
Clinical presentation
Symptoms
- General malaise/tiredness - often duet iron deficiency anaemia
Signs
- Dermatitis herpetiformis - itchy blisters caused by subepithelial IgA deposition
Investigations
- Serology (IgA anti-TTG) use to screen who needs a biopsy
- Patients need to be consuming gluten for 4-6 weeks before biopsy/serology
Management
- Lifelong adherence to a gluten free diet
Complications
- Refractory coeliac disease can progress to T-cell lymphoma