A parasitic infection caused by the intestinal nematode Strongyloides stercoralis.
Aetiology
Causative Organisms
Strongyloides stercoralis

Pathophysiology
The infection occurs via the fecal–oral route.
Life Cycle Stages
- Filariform larvae in soil penetrate human skin
- Enter bloodstream → migrate to lungs
- Ascend bronchial tree → swallowed
- Mature into adult female worms in small intestine
- Eggs hatch into rhabditiform larvae in intestine
- Larvae are:
- Excreted in stool, or
- Cause autoinfection by penetrating intestinal mucosa or perianal skin
Autoinfection explains chronic infection and hyperinfection syndrome.

Pathogenesis
Stage | Pathology |
Skin penetration | Local inflammation |
Pulmonary migration | Eosinophilic pneumonitis |
Intestinal phase | Mucosal inflammation |
Autoinfection | Continuous parasite cycle |
Hyperinfection | Massive dissemination in immunocompromised patients |
Severe disseminated disease is common in patients with:
- Corticosteroid therapy
- Organ transplantation
- Hematologic malignancy
- Human Immunodeficiency Virus Infection (less common but possible)
Clinical Presentation
Investigations
Test | Findings |
Stool microscopy | Rhabditiform larvae |
Stool concentration techniques | Increased detection |
Agar plate culture | Larval tracks |
Serology (ELISA) | Detect antibodies |
CBC | Eosinophilia |
Sputum exam | Larvae in hyperinfection |
Important point:
Eggs are rarely seen in stool because they hatch in the intestine.
Management
First-Line
Drug | Dose |
Ivermectin | 200 µg/kg/day for 1-2 days |
Albendazole | 400 mg 2x1 for 7 days |
