Chronic disease caused by Mycobacterium tuberculosis (generally)
Aetiology
- M. tb is the main causative organism in humans – rod-shaped gram-positive bacillus, acid fast
- M. bovis can jump the species barrier from cattle to humans
Risk factors
- Immunosuppression - virulent MTB or atypical infection
Pathophysiology
- Type IV hypersensitivity - granulomas with necrosis
- Pathogen transmitted via aerosol route and reaches alveoli
- Pathogen phagocytosed in alveoli and carried to hilar lymph nodes → immune activation
- This leads to a granulomatous response in the nodes, with caseous necrosis occurring in the granulomas
- The infection is either cleared completely or will become latent (LTBI)
- In a few cases the infection is overwhelming and spreads throughout the body, setting up many foci of infection (e.g. miliary TB) - extra-pulmonary TB
Clinical presentation
Pulmonary features
- 90% present with pulmonary features only
Extrapulmonary features (10%)
- Range of other organ-specific symptoms
Investigations
Active TB
- CXR
- Shadows, lesions, consolidation
- Ghon focus in periphery of mid zone of lung - primary site of infection
- Bilateral hilar lymphadenopathy
- ‘Miliary shadowing’ = miliary TB
- Samples e.g. sputum, pus, or a tissue biopsy
- 3 separate sputum samples in pulmonary TB (including one early morning sample)
- Can do broncoscopy and lavage or gastric washings (rarely required)
- Ziehl-Neelson stain - tests for AFB, rapid (24h)
- Histology - granuloma with central caseous necrosis
Latent TB
Management
Active TB
- Rifampicin, Isoniazid, Pyrazinamide and Ethambutol for 4 months
- Rifampicin and Isoniazid for a further 2 months
Latent TB
- Rifampicin and Isoniazid for 3 months
- OR Isoniazid for 6 months
- SGOT/SGPT three fold with nausea and vomiting